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development of IAA was regarded as very likely on account of oxidase deamination of NMT (Barker et al., 1980), but was later on set up for being also in part by immediate deamination by MAO (Barker et al., 1982). Pretreatment With all the MAO-I iproniazid in rat complete Mind homogenates inhibited IAA development by 83%, NMT and DMT-NO formation ha